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viagra® improves erection and the possibility of successful intercourse. The drug works for 36 hours. The effect appears within 16 minutes after taking the drug in the presence of sexual arousal.
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viagra® is an effective, reversible, selectiveth inhibitor of specific phosphodiesterase type 5 (PDE5) cyclic guanosine monophosphate (cGMP)Order viagra
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These vascular effects may be accompanied by inhibition of the activity of the afferent nerves in the urinary bladder and relaxation of the smooth muscles of the prostate and bladder.
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In vitro studies have shown that viagra® is a selective PDE5 inhibitor. PDE5 is an enzyme found in the smooth muscles of the corpus cavernosum, in the smooth muscles of the vessels of the internal organs, in skeletal muscles, platelets, kidneys, lungs and cerebellum. The action of viagra® on PDE5 is more active than on other phosphodiesterases. viagra® is 10,000 times more potent for PDE5 than for PDE1, PDE2, PDE4 and PDE7, which are localized in the heart, brain, blood vessels, liver, leukocytes, skeletal muscles and other organs. viagra® is 10,000 times more active in blocking PDE5 than PDE3, an enzyme found in the heart and blood vessels. This selectivity for PDE5 over PDE3 is important because PDE3 is an enzyme involved in cardiac muscle contraction.

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In addition, viagra® is approximately 700 times more active against PDE5 than against PDE6, an enzyme found in the retina of the eye and responsible for phototransmission. viagra® also exhibits an effect 9,000 times more potent against PDE5 compared to its effect on PDE8, PDE9 and PDE10, and 14 times more potent against PDE5 than PDE11. Tissue distribution and physiological effects of inhibition of PDE8 - PDE11 have not yet been clarified.

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viagra® in healthy individuals does not cause significant changes in systolic and diastolic pressure in comparison with placebo in the supine position (the average maximum decrease is 1.6 / 0.8 mm Hg, respectively) and in the standing position (the average maximum decrease is 0.2 / 4.6 mm Hg. Art., respectively). viagra® does not cause significant changes in heart rate.

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viagra® does not cause changes in color recognition (blue / green), which is explained by its low affinity for PDE6 compared to PDE5. In addition, the effect of viagra® on visual acuity, electroretinogram, intraocular pressure and pupil size is not observed.

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Three studies were conducted on male patients in order to assess the potential effect on spermatogenesis of daily intake of viagra® 10 mg (one 6-month study) and 20 mg (one 6-month and one 9-month study). None of the studies observed an undesirable effect on sperm morphology and motility. Two studies found a decrease in sperm concentration compared to placebo. In a 9-month study, decreased sperm concentration was associated with higher ejaculation rates.

Ejaculation rates were not evaluated in the 6-month studies. In addition, there was no undesirable effect on the average concentration of sex hormones, testosterone, luteinizing hormone and follicle-stimulating hormone when taking viagra® 10 or 20 mg, compared with placebo.